Saab

Describes motivation process for creativity with emphasis on intrinsic motivation by Corey K Katir

legalweek

Allen & Overy (A&O) has taken a lead role as General Motors (GM) and Spyker reached an agreement for the purchase of Saab Automobile yesterday (26 January) after months of discussions. The magic circle firm advised Spyker on the deal, which has been agreed at a cut-price value of $400m (247m) in cash and shares, but will see the small Dutch auto maker save the Saab brand from disappearing after GM announced plans to wind down operations late last year.

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Mayer Brown has taken the lead role for Beijing Automotive Industry Holding (BAIC) on the purchase of Saab assets and intellectual property rights from General Motors. The deal, valued at SEK1.4bn (119m), could stave off bankruptcy for Swedish car maker Saab while GM continues to search for a buyer, following the failed takeover attempt by Swedish car maker Koenigsegg in the autumn.

legalweek

Skadden Arps Slate Meagher & Flom has advised Sweden’s Koenigsegg on its acquisition of General Motors’ loss-making brand Saab. The deal, which was backed by Norwegian and US investors, saw Skadden field a team including City corporate finance partner Pranav Trivedi.

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Spyker, maker of hand-built exotic cars, is selling that business to a Russian billionaire, while it concentrates on reviving Swedish premium carmaker Saab.

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A final batch of 26 Saab 9-3 convertibles has arrived for sale in the UK.

In this edition of The Fast Lane, David Booth looks at the strung-out drama that is Saab, and points out why it probably isn’t worth saving.

After 60 years of quirkiness and innovation, Saab is teetering on the brink of existence

Oligodendrocytes, the myelin-forming glial cells of the central nervous system, maintain long-term axonal integrity. However, the underlying support mechanisms are not understood. Here we identify a metabolic component of axonaglia interactions by generating conditional Cox10 (protoheme IX farnesyltransferase) mutant mice, in which oligodendrocytes and Schwann cells fail to assemble stable mitochondrial cytochrome c oxidase (COX, also known as mitochondrial complex IV). In the peripheral nervous system, Cox10 conditional mutants exhibit severe neuropathy with dysmyelination, abnormal Remak bundles, muscle atrophy and paralysis. Notably, perturbing mitochondrial respiration did not cause glial cell death. In the adult central nervous system, we found no signs of demyelination, axonal degeneration or secondary inflammation. Unlike cultured oligodendrocytes, which are sensitive to COX inhibitors, post-myelination oligodendrocytes survive well in the absence of COX activity. More importantly, by in vivo magnetic resonance spectroscopy, brain lactate concentrations in mutants were increased compared with controls, but were detectable only in mice exposed to volatile anaesthetics. This indicates that aerobic glycolysis products derived from oligodendrocytes are rapidly metabolized within white matter tracts. Because myelinated axons can use lactate when energy-deprived, our findings suggest a model in which axonaglia metabolic coupling serves a physiological function.

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